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@denielcramer: Perfect life hack for when you‘re sharing a board with a homie 😂 #skateboard #skatelife #fyp

Deniel Cramer
Deniel Cramer
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Region: DE
Sunday 04 June 2023 13:25:15 GMT
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Comments

mrwaltersxm
MrWalter :
Presque Seb un peu non ?
2023-06-05 21:09:28
17
alexlancho2
Alex Lancho 🥨 :
They need to nerf that
2023-06-05 18:46:57
14
ghosttktok
Ghost :
They need to patch this
2023-06-05 04:11:38
1548
ash.ley_80
☆ :
Nice one tho I never thought about this one😁
2023-06-04 17:39:17
229
ahmeeet10
SUPERSTARS<3 :
Warte das ist so smart Digga
2023-06-04 21:25:38
379
maxi24.10_
FRUCHTEI :
Unge ?
2023-06-04 14:15:25
119
headshot10105
Headshot10104 :
Nah bro just split it in half 😂😂
2023-09-19 04:40:29
23
4nn1080
Nv4 :
bro just found a travel hack
2025-03-16 21:23:41
22
whereskarmadog
whereskarmadog :
Lol man I forgot about this! Good times.
2023-06-06 02:51:53
2
myassholeruptured
Godin :
nah my bushings are split
2023-06-06 12:01:09
5
nido_7aleeb
Nido 7aleeb🚦 :
Wasnt this in sk8? Reki and langa moment
2023-07-10 19:23:09
6
warnichtsanderesmehrfrei
BMO :
Also ich würde meinem dude sagen er soll sein Board zum Skaten auch mitbringen 🤔
2023-06-05 08:53:13
49
chicoelefante0
lalonganiza :
banda lo intente y ya arreglaron el bug :(
2023-06-06 18:48:26
224
magui.sousa_
megui :
como eu nunca pensei nisso
2023-08-18 08:12:15
1
olivarius4
olivarius💀 :
Brat znalazl sposob na rotacje
2023-06-08 23:51:00
62
_noowey_
n𝄞wey :
C’est à lisbon ?
2023-06-08 04:49:24
3
sentiasa_betul
юля всегда права :
комментирую видео со скейтом #55
2023-07-13 03:20:06
4
davidefilipporossi
Daaav🍃 :
Yes bro the same😂
2023-06-05 05:44:41
2
nick.ept
Nick :
hat jemand Bock Mal skaten zu gehen (baden Württemberg)
2023-08-03 20:01:21
1
rumpsportugal
RUMPS SKATEBOARD :
Lisbon 🔥👑
2024-05-31 09:33:23
1
vascomvzd_
Deli :
Are you in portugal
2023-06-04 14:01:14
3
snkyb_ardo
Sneakyb :
Lost his board? 🤣
2023-06-06 09:09:49
2
blako1
Blako1 :
No way you are skating with 4s
2023-10-05 17:00:49
3
imchoforo
imchoforo :
Omg i would do this with my cousin 💀 we thought we were geniuses
2023-06-05 18:03:16
2
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Other Videos

hEDS has traditionally been classified as a connective tissue disorder defined by joint hypermobility. This framing never fully explained why hEDS so often comes with immune dysfunction, dysautonomia, and chronic fatigue. New genetic research is reframing it as a multi-system condition. A 2025 study by Gensemer et al. in iScience (MUSC, Norris lab) performed whole-exome sequencing on 200 hEDS patients and identified a recurrent variant in KLK15, a gene active in both connective tissue and immune tissue. A knock-in mouse with the variant developed connective tissue abnormalities in tendons and heart valves alongside dysregulated cytokine signaling. A separate 2026 study by Shirvani et al. in Genes found hEDS-associated variants enriched across three systems: collagen biosynthesis, HLA/immune pathways, and mitochondrial function. KLK15 is described as a contributor, not a confirmed single cause. The variant was found in roughly a third of patients, and hEDS remains genetically complex. This research does not yet enable a genetic test for hEDS, and the 2017 clinical criteria remain the standard for diagnosis. The multi-system genetic paper used machine learning on a single cohort and needs replication. I’m not a doctor, just sharing research I find interesting. Sources: Gensemer et al. (2025). iScience. DOI: 10.1016/j.isci.2025.113343 Shirvani et al. (2026). Genes. DOI: 10.3390/genes17020211 #heds #ehlersdanlos #chronicillness #hypermobileehlersdanlossyndrome
hEDS has traditionally been classified as a connective tissue disorder defined by joint hypermobility. This framing never fully explained why hEDS so often comes with immune dysfunction, dysautonomia, and chronic fatigue. New genetic research is reframing it as a multi-system condition. A 2025 study by Gensemer et al. in iScience (MUSC, Norris lab) performed whole-exome sequencing on 200 hEDS patients and identified a recurrent variant in KLK15, a gene active in both connective tissue and immune tissue. A knock-in mouse with the variant developed connective tissue abnormalities in tendons and heart valves alongside dysregulated cytokine signaling. A separate 2026 study by Shirvani et al. in Genes found hEDS-associated variants enriched across three systems: collagen biosynthesis, HLA/immune pathways, and mitochondrial function. KLK15 is described as a contributor, not a confirmed single cause. The variant was found in roughly a third of patients, and hEDS remains genetically complex. This research does not yet enable a genetic test for hEDS, and the 2017 clinical criteria remain the standard for diagnosis. The multi-system genetic paper used machine learning on a single cohort and needs replication. I’m not a doctor, just sharing research I find interesting. Sources: Gensemer et al. (2025). iScience. DOI: 10.1016/j.isci.2025.113343 Shirvani et al. (2026). Genes. DOI: 10.3390/genes17020211 #heds #ehlersdanlos #chronicillness #hypermobileehlersdanlossyndrome


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