@pleb_kruse: This mitochondriac perspective helps explains why parabiosis studies in the literature have shown why young blood shows so many health benefits in disease states. Blood that is younger from a circadian standpoint, has the ability to clear these fragmented heme-based molecules from damaged cells. I have hypothesized in my Patreon blog series that RBCS may attenuate CpG-induced inflammation in multiple organs through direct scavenging of CpG-containing DNA released from damaged cells. How does our blood plasma link to this perspective Uncle Jack? The innate immune system appears to use TLR9 on our blood cells for detecting unmethylated CpG dinucleotides which are liberated during the blue light hazard and nEMF exposure associated with mental disease that leads to suicide. The CpG Islands help tell the immune system how much damage has been done by pathogens and how to react. Why? CpG islands in pathogens vary compared to our own from our DNA/RNA and mtDNA. CpG islands are relatively common in bacterial and viral genomes but are highly methylated and uncommon in vertebrate genomes. People forget mitochondria have a bacterial lineage and this is why mitochondrial damage leads to a hypermethylated state in our CNS and PNS. #doctor #usa #sunset #typusa
