@kuuchan_.m: 激ハマリ中のげきやくちゅうどく🩵🎀#イコラブ #イコールラブ #げきやくちゅうどく

Mikuru🦋☁️

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Region: JP

Tuesday 21 April 2026 12:35:51 GMT

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Comments

はると！！！🦋☁️ :

歌めっちゃ上手！！！

2026-04-21 13:48:59

3

オリオントールcamp🏕️ :

くーちゃんの声めっちゃ好き！高音に上がるところとかめっちゃ耳心地がイイ❣️

2026-04-21 13:23:16

2

勇次郎 :

歌声もええな😆😆

2026-04-21 14:09:28

1

やかん :

くーちゃん中毒♡😊

2026-04-22 11:14:33

1

山二郎 :

くーちゃんの歌声が1番好き🥰

2026-04-21 13:24:50

1

ぱぴたん🐰💜 :

上手すぎ😍

2026-04-21 12:58:54

1

のんちゃん(野元空・エレエネ生涯推し)☀️ :

くーちゃんの歌声好きすぎる🥰

2026-04-21 20:03:45

1

BOB naga :

やっぱあいかわらず歌うまーい❕

2026-04-22 13:42:27

1

たろぽん :

大好きすぎます❤️

2026-04-21 14:23:32

1

ツァラトラAp-B :

俺も好きな激安昼食、くーちゃんも好きで嬉しいな😂

2026-04-21 15:28:16

1

user7345433640587 :

素晴らしい

2026-04-21 14:22:15

1

さとる :

上手い！昔からくーちゃんの歌声がめっちゃ好きなんだよな〜💗💗

2026-04-22 05:35:08

1

Natata :

うまい

2026-04-21 17:01:43

1

あ :

ちかい！かわいい

2026-04-21 15:06:25

1

「朩ﾟ朩ﾟ」🅿️🦟🐷…匕ﾟ力卜冫…㌧㌧ :

久しぶりの歌声！いい声

2026-04-21 13:13:04

1

ともちん(ともき) :

歌上手いです

2026-04-21 22:57:38

1

ふるまる :

かわいい💕

2026-04-21 15:02:59

1

き.の.こ. :

素敵です🥰💞

2026-04-21 12:42:49

1

やごろ〜 :

やっぱ好きな声やわー✨歌もっと聞きたい！

2026-04-21 13:42:24

1

マーシー沖縄 :

久しぶり見たけど。可愛いさー

2026-04-21 21:55:25

1

たいくーん :

相変わらずすごい歌唱力ですね🥺

2026-04-21 13:28:59

1

へほほのポポポ :

うますぎるよ！！！

2026-04-21 12:42:01

1

マ－ミ－🧡🦉🌸🧚‍♂️ :

キタキタキタキタみくるちゃんボイス🩵声量やっべぇゾウパオーン🐘容姿かわいすぎ優勝マジマジマジンガーやっべぇゾウメロメロメロい🍈😊

2026-04-21 18:36:11

1

토모야(ともや) :

お疲れ〜😍最後の「ムズ」って言ってるくーちゃんオモロい👍頑張って🔥

2026-04-21 12:42:41

1

たいぴー🐺🌟 :

歌うまいし可愛いな

2026-04-21 12:37:37

1

To see more videos from user @kuuchan_.m, please go to the Tikwm homepage.

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hEDS has traditionally been classified as a connective tissue disorder defined by joint hypermobility. This framing never fully explained why hEDS so often comes with immune dysfunction, dysautonomia, and chronic fatigue. New genetic research is reframing it as a multi-system condition. A 2025 study by Gensemer et al. in iScience (MUSC, Norris lab) performed whole-exome sequencing on 200 hEDS patients and identified a recurrent variant in KLK15, a gene active in both connective tissue and immune tissue. A knock-in mouse with the variant developed connective tissue abnormalities in tendons and heart valves alongside dysregulated cytokine signaling. A separate 2026 study by Shirvani et al. in Genes found hEDS-associated variants enriched across three systems: collagen biosynthesis, HLA/immune pathways, and mitochondrial function. KLK15 is described as a contributor, not a confirmed single cause. The variant was found in roughly a third of patients, and hEDS remains genetically complex. This research does not yet enable a genetic test for hEDS, and the 2017 clinical criteria remain the standard for diagnosis. The multi-system genetic paper used machine learning on a single cohort and needs replication. I’m not a doctor, just sharing research I find interesting. Sources: Gensemer et al. (2025). iScience. DOI: 10.1016/j.isci.2025.113343 Shirvani et al. (2026). Genes. DOI: 10.3390/genes17020211 #heds #ehlersdanlos #chronicillness #hypermobileehlersdanlossyndrome

hEDS has traditionally been classified as a connective tissue disorder defined by joint hypermobility. This framing never fully explained why hEDS so often comes with immune dysfunction, dysautonomia, and chronic fatigue. New genetic research is reframing it as a multi-system condition. A 2025 study by Gensemer et al. in iScience (MUSC, Norris lab) performed whole-exome sequencing on 200 hEDS patients and identified a recurrent variant in KLK15, a gene active in both connective tissue and immune tissue. A knock-in mouse with the variant developed connective tissue abnormalities in tendons and heart valves alongside dysregulated cytokine signaling. A separate 2026 study by Shirvani et al. in Genes found hEDS-associated variants enriched across three systems: collagen biosynthesis, HLA/immune pathways, and mitochondrial function. KLK15 is described as a contributor, not a confirmed single cause. The variant was found in roughly a third of patients, and hEDS remains genetically complex. This research does not yet enable a genetic test for hEDS, and the 2017 clinical criteria remain the standard for diagnosis. The multi-system genetic paper used machine learning on a single cohort and needs replication. I’m not a doctor, just sharing research I find interesting. Sources: Gensemer et al. (2025). iScience. DOI: 10.1016/j.isci.2025.113343 Shirvani et al. (2026). Genes. DOI: 10.3390/genes17020211 #heds #ehlersdanlos #chronicillness #hypermobileehlersdanlossyndrome

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tum mujhe dara kyun rahe ho? 😈🔥 Video aur song kaisa laga? Sab ko sahi laga? 👀👇 #WindBreaker #Suho #AnimeEdit #Editz #FYP

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