@passiondufoot02: La Belgique tenue EN ÉCHEC par l'Iran 🔥 #worldcup2026 #iran #belgium #pourtoi

Passion du foot ❤️‍🔥
Passion du foot ❤️‍🔥
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Sunday 21 June 2026 22:48:53 GMT
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lesaintpatrolier241
Le saint Patrolier gaboma🇬🇦 :
félicitations aux gardiens
2026-06-22 01:47:57
0
adjilebamba1
Adjile Bamba :
Les gardiens étaient chaud
2026-06-21 23:06:21
2
la.pulga8493
BULMA :
Courthois a part Buffon qui est devant cette légende ?? Le nombre d’arrêt décisives qu’il a effectué c’est Hallucinant !!
2026-06-22 02:35:35
0
lulamimichel
Tel il est, Tel je suis aussi :
Quels gardiens héroïques de deux côtés!!
2026-06-22 00:13:45
1
ousmanesanogo3966
ousmanesanogo3966 :
😁😁😁
2026-06-21 23:06:32
0
nassarotsman
نصار محب للبنان :
❤️❤️❤️
2026-06-21 23:12:46
0
morro.seybouhuk
Morro Seybouhuk :
🥰🥰🥰
2026-06-21 23:58:10
0
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Hello everyone, I’m Dr. PK, and today we are talking about gout — one of the most painful and misunderstood forms of arthritis. Many people think gout is just from eating too much meat or drinking alcohol. That is only part of the story. In this video, I’ll explain: what gout really is, the pathophysiology in simple language, who gets it, how to prevent attacks, and the best treatment options for both sudden flares and long-term control. Stay till the end, because I will also explain why treating only the pain is not enough. Part 1: What is gout? (0:45–1:40) Gout is a type of inflammatory arthritis caused by uric acid crystal deposition in the joints. Normally, uric acid is a waste product formed when the body breaks down purines. Purines come from: your own body’s natural cell turnover, ad certain foods and drinks. Usually, uric acid dissolves in the blood, passes through the kidneys, and leaves the body in urine. But when uric acid builds up too much in the blood, it can form needle-shaped monosodium urate crystals. These crystals deposit in joints, soft tissues, and sometimes kidneys. That is what causes gout. The classic site is the big toe, but gout can also affect: ankle, foot, knee, wrist, fingers, and elbow. Part 2: Pathophysiology — what is happening inside the body? (1:40–3:20) Let’s make this simple. Step 1: Uric acid goes up Gout usually starts when the blood urate level becomes high enough that crystals can form. Crystals tend to form once urate exceeds the saturation threshold, roughly around 6.8 mg/dL, which is why treatment aims to keep levels below that range. NICE recommends aiming for serum urate below 6 mg/dL, and for some patients with tophi or ongoing flares, below 5 mg/dL may be considered. Step 2: Crystals deposit in the joint These crystals sit silently in the joint for a while. A person may not even know they are there. Step 3: The immune system attacks Then suddenly, the immune system recognizes these crystals as a danger signal. White blood cells rush in and release inflammatory chemicals. That is why a gout attack becomes: sudden, red, hot, swollen, and intensely painful. Step 4: Recurrent inflammation causes damage If gout keeps hap
Hello everyone, I’m Dr. PK, and today we are talking about gout — one of the most painful and misunderstood forms of arthritis. Many people think gout is just from eating too much meat or drinking alcohol. That is only part of the story. In this video, I’ll explain: what gout really is, the pathophysiology in simple language, who gets it, how to prevent attacks, and the best treatment options for both sudden flares and long-term control. Stay till the end, because I will also explain why treating only the pain is not enough. Part 1: What is gout? (0:45–1:40) Gout is a type of inflammatory arthritis caused by uric acid crystal deposition in the joints. Normally, uric acid is a waste product formed when the body breaks down purines. Purines come from: your own body’s natural cell turnover, ad certain foods and drinks. Usually, uric acid dissolves in the blood, passes through the kidneys, and leaves the body in urine. But when uric acid builds up too much in the blood, it can form needle-shaped monosodium urate crystals. These crystals deposit in joints, soft tissues, and sometimes kidneys. That is what causes gout. The classic site is the big toe, but gout can also affect: ankle, foot, knee, wrist, fingers, and elbow. Part 2: Pathophysiology — what is happening inside the body? (1:40–3:20) Let’s make this simple. Step 1: Uric acid goes up Gout usually starts when the blood urate level becomes high enough that crystals can form. Crystals tend to form once urate exceeds the saturation threshold, roughly around 6.8 mg/dL, which is why treatment aims to keep levels below that range. NICE recommends aiming for serum urate below 6 mg/dL, and for some patients with tophi or ongoing flares, below 5 mg/dL may be considered. Step 2: Crystals deposit in the joint These crystals sit silently in the joint for a while. A person may not even know they are there. Step 3: The immune system attacks Then suddenly, the immune system recognizes these crystals as a danger signal. White blood cells rush in and release inflammatory chemicals. That is why a gout attack becomes: sudden, red, hot, swollen, and intensely painful. Step 4: Recurrent inflammation causes damage If gout keeps hap

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