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@l.8ky: . #foryou #fyp #viral #موصل #tiktok

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حـَسـن
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Sunday 28 June 2026 17:28:49 GMT
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ua_541
حسوني ابن ساجد :
هوه حسون واني حسون وين اكو عداله 🥲😅
2026-06-28 21:23:03
12
zhoor2468
𝙽 🦢 :
يمه شون جماللل❤️❤️
2026-06-28 18:10:46
8
.rafal43
RN :
باع التعاليق
2026-06-29 15:35:42
5
2_ms_5
. :
اشلون اسوي شعري هيج
2026-06-28 21:59:52
1
.172940
𝑴𝒐𝒘𝒂𝒇𝒂𝒒 𝑱𝒂𝒔𝒔𝒆𝒎 :
شلونن كيككك هاذ😍💙
2026-06-29 23:37:50
0
sagedcr1
سـاجـد🇧🇷 :
ياابه صدگ تشبهنييي..؟
2026-07-28 03:23:48
0
j19x4
انس :
حبيب قلبي❤️
2026-06-28 17:41:16
1
h.sa_nn
حسن✨ :
تخبل حبيبي✨
2026-06-28 21:26:41
0
youssef.alabbasi6
Youssef Al-Abbasi :
ربي يحفظك❤️
2026-06-28 17:57:11
1
cx.a26
اسماعيل يحيى الطائي 🤍 :
حبيب القلب 🤍🔥
2026-06-28 21:37:21
1
muntazir.adil
Muntazir Adil :
تخبل حبي🥰🥰
2026-06-28 22:22:51
0
userytv0raadw
Abbas 💎 :
الحبيب ❤️
2026-06-28 18:32:16
0
.2032324
حمودي الكويري 2 :
نورت🌹
2026-06-28 18:32:28
0
00xx166
نعمان || NuemaN 💸💎 :
ححبيبي🙂‍↕️
2026-06-28 18:18:47
0
s3.9m
حمَد :
كل يوم يزيد جمالللك
2026-06-28 22:05:40
1
o5d_i
الـنعـيمـي515🍃🇮🇶® :
تخبل اخويه🔥💕
2026-06-28 21:09:33
0
ay1_c
أيـــمــن∆ :
جمالك اخـي❤️
2026-06-28 22:15:08
0
ali8_h28
Ali 🎞️ :
روحي❤️❤️❤️
2026-06-28 17:36:38
0
al.ahad273
عَـــــمورري♥️👍🏿. :
حسوني لأنيق ♥️😄.
2026-06-28 22:34:47
1
dewhurstfyan
Youssef :
الله يحفضك انشالله ❤️❤️
2026-06-28 17:43:15
1
8h762
نويره :
دباع الجمال 🤏😫
2026-06-28 19:45:09
4
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Winter is coming 👀 @Heavenly Heat Saunas Shop the huge Labor Day Sale happening before it ends on September 9th. Hit the link in my bio and use code CADEN for even more savings 💪🏻🔥
Winter is coming 👀 @Heavenly Heat Saunas Shop the huge Labor Day Sale happening before it ends on September 9th. Hit the link in my bio and use code CADEN for even more savings 💪🏻🔥
4 WAZIIFY YAD KER LEHN ll repost request 🥹❤️‍🩹 ll ᴅᴇᴀʀ ᴛɪᴋᴛᴏᴋ ᴛᴇᴀᴍ ᴅᴏɴ'ᴛ ᴜɴᴅᴇʀ ʀᴇᴠɪᴇᴡ ᴍʏ ᴠɪᴅᴇᴏ #peerajmalrazaqadri #viralvideos #islamic #foryoupage #growmyaccount
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متابعة لا يك اعادة نشر يا غوالي 🤝
متابعة لا يك اعادة نشر يا غوالي 🤝
unfreeze my account #fyp #foryoupage #1millionaudition #aesthtic #poetrystatus
unfreeze my account #fyp #foryoupage #1millionaudition #aesthtic #poetrystatus


hEDS has traditionally been classified as a connective tissue disorder defined by joint hypermobility. This framing never fully explained why hEDS so often comes with immune dysfunction, dysautonomia, and chronic fatigue. New genetic research is reframing it as a multi-system condition. A 2025 study by Gensemer et al. in iScience (MUSC, Norris lab) performed whole-exome sequencing on 200 hEDS patients and identified a recurrent variant in KLK15, a gene active in both connective tissue and immune tissue. A knock-in mouse with the variant developed connective tissue abnormalities in tendons and heart valves alongside dysregulated cytokine signaling. A separate 2026 study by Shirvani et al. in Genes found hEDS-associated variants enriched across three systems: collagen biosynthesis, HLA/immune pathways, and mitochondrial function. KLK15 is described as a contributor, not a confirmed single cause. The variant was found in roughly a third of patients, and hEDS remains genetically complex. This research does not yet enable a genetic test for hEDS, and the 2017 clinical criteria remain the standard for diagnosis. The multi-system genetic paper used machine learning on a single cohort and needs replication. I’m not a doctor, just sharing research I find interesting. Sources: Gensemer et al. (2025). iScience. DOI: 10.1016/j.isci.2025.113343 Shirvani et al. (2026). Genes. DOI: 10.3390/genes17020211 #heds #ehlersdanlos #chronicillness #hypermobileehlersdanlossyndrome
hEDS has traditionally been classified as a connective tissue disorder defined by joint hypermobility. This framing never fully explained why hEDS so often comes with immune dysfunction, dysautonomia, and chronic fatigue. New genetic research is reframing it as a multi-system condition. A 2025 study by Gensemer et al. in iScience (MUSC, Norris lab) performed whole-exome sequencing on 200 hEDS patients and identified a recurrent variant in KLK15, a gene active in both connective tissue and immune tissue. A knock-in mouse with the variant developed connective tissue abnormalities in tendons and heart valves alongside dysregulated cytokine signaling. A separate 2026 study by Shirvani et al. in Genes found hEDS-associated variants enriched across three systems: collagen biosynthesis, HLA/immune pathways, and mitochondrial function. KLK15 is described as a contributor, not a confirmed single cause. The variant was found in roughly a third of patients, and hEDS remains genetically complex. This research does not yet enable a genetic test for hEDS, and the 2017 clinical criteria remain the standard for diagnosis. The multi-system genetic paper used machine learning on a single cohort and needs replication. I’m not a doctor, just sharing research I find interesting. Sources: Gensemer et al. (2025). iScience. DOI: 10.1016/j.isci.2025.113343 Shirvani et al. (2026). Genes. DOI: 10.3390/genes17020211 #heds #ehlersdanlos #chronicillness #hypermobileehlersdanlossyndrome

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