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חמודה 🦅
חמודה 🦅
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Monday 10 August 2026 10:40:34 GMT
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Type 1 diabetes begins when immune cells wipe out insulin producing beta cells. Glucose enters the bloodstream after a meal, raising blood sugar levels. The pancreas detects this rise, and its beta cells release insulin. Insulin travels through the blood and binds to insulin receptors on body cells. This activates glucose transporters on the cell membrane, allowing glucose to enter the cells. As glucose moves into cells and is used for energy, blood sugar levels return to normal. In Type 1 diabetes, the immune system mistakenly targets and destroys the pancreatic beta cells. As beta cells are damaged, insulin production rapidly decreases. Without insulin, glucose cannot enter cells, and glucose accumulates in the bloodstream. Cells become energy-deprived despite high circulating glucose. Blood sugar continues to rise, leading to hyperglycemia. This causes symptoms such as excessive thirst, frequent urination, fatigue, and unintentional weight loss. External insulin becomes necessary to regulate blood glucose.
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