@anahi0230: Que vivan los hombres que envían flores!!! #novios #pareja #amor

Anahita 🍒
Anahita 🍒
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Region: EC
Sunday 16 August 2026 00:53:16 GMT
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whoismaurg01
𝓐𝓵𝓮𝔁𝓪𝓷𝓭𝓮𝓻 ♣️ :
Tienes razón, ya que no somos a quienes nos dan ese tipo de detalles, generalmente no recibimos cosas así, la sociedad tiene tan metido en la cabeza que solo hay que proveer
2026-08-16 01:44:16
17
luis_ssj111
LUIS_SSJ :
Cuando den
2026-08-16 22:39:38
0
smith.smith666
smith smith :
No puedes porque no la experimentamos pues
2026-08-16 21:14:01
1
cristian20075
Cristian :
ya 19 años y sólo una mujer me ha dado cartas y peluche y es tan lindo tanto que no sabes ni que decir yo personalmente me comenze a casi llorar.
2026-08-16 14:28:53
0
jhoansebastian2025
Sebas L. D. U 🥁🤍 :
Pues la vrd nose nunca he sentido nd pq en mis 20 años de edad nunca me han regalado ni un chicle de 5 ctv jaja pero soy muy feliz q las mujeres si puedan sentir ese sentimiento pq las mujeres lo es todo para este mundo q Dios les bendiga amén 😢❤️‍🩹
2026-08-18 05:24:14
0
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High testosterone in women does NOT automatically mean PCOS. When I see elevated testosterone on a blood test, I’m much more interested in why it is elevated and where those androgens are coming from. Women naturally produce androgens. They’re produced predominantly by the ovaries and adrenal glands, with additional peripheral conversion occurring in other tissues. In the ovaries, LH stimulates theca cells to produce androgens. But there’s another important driver that can amplify this process: insulin. When insulin is persistently elevated, it can act alongside LH to increase ovarian androgen production. This is one reason hyperinsulinaemia can be such an important driver of androgen excess in some women with PCOS. But insulin can affect testosterone from another direction too. Higher insulin can suppress hepatic production of SHBG — sex hormone-binding globulin. SHBG binds circulating testosterone, so when SHBG falls, the proportion of testosterone that is free and biologically available can increase. This means you can experience clinical signs of androgen excess without having an extraordinarily high total testosterone result. And PCOS isn’t the only explanation. The adrenal glands also produce androgens, including DHEA and DHEAS. If these are elevated, I start thinking about whether there may be a greater adrenal contribution. Medications can influence androgen levels, and significant or rapidly developing androgen excess can require further medical investigation to exclude less common ovarian or adrenal causes. This is why I don’t interpret testosterone in isolation. I want to understand total testosterone, SHBG, calculated free testosterone/free androgen index, DHEA/DHEAS, alongside menstrual patterns, ovulation, clinical symptoms and metabolic markers such as insulin. Because “high testosterone” is a finding. It isn’t the diagnosis. The more important clinical question is: what is driving it? If you’ve been told you have elevated testosterone or you’re investigating PCOS, this is something we can work through at Nourishing Apothecary. Danielle and Toni both work extensively with women’s hormonal and metabolic health. You can book a free discovery call to discuss your case and determine the right practitioner for you at nourishingapothecary.com. #HighTestosterone #PCOS #Androgens #WomensHormones #HormoneHealth
High testosterone in women does NOT automatically mean PCOS. When I see elevated testosterone on a blood test, I’m much more interested in why it is elevated and where those androgens are coming from. Women naturally produce androgens. They’re produced predominantly by the ovaries and adrenal glands, with additional peripheral conversion occurring in other tissues. In the ovaries, LH stimulates theca cells to produce androgens. But there’s another important driver that can amplify this process: insulin. When insulin is persistently elevated, it can act alongside LH to increase ovarian androgen production. This is one reason hyperinsulinaemia can be such an important driver of androgen excess in some women with PCOS. But insulin can affect testosterone from another direction too. Higher insulin can suppress hepatic production of SHBG — sex hormone-binding globulin. SHBG binds circulating testosterone, so when SHBG falls, the proportion of testosterone that is free and biologically available can increase. This means you can experience clinical signs of androgen excess without having an extraordinarily high total testosterone result. And PCOS isn’t the only explanation. The adrenal glands also produce androgens, including DHEA and DHEAS. If these are elevated, I start thinking about whether there may be a greater adrenal contribution. Medications can influence androgen levels, and significant or rapidly developing androgen excess can require further medical investigation to exclude less common ovarian or adrenal causes. This is why I don’t interpret testosterone in isolation. I want to understand total testosterone, SHBG, calculated free testosterone/free androgen index, DHEA/DHEAS, alongside menstrual patterns, ovulation, clinical symptoms and metabolic markers such as insulin. Because “high testosterone” is a finding. It isn’t the diagnosis. The more important clinical question is: what is driving it? If you’ve been told you have elevated testosterone or you’re investigating PCOS, this is something we can work through at Nourishing Apothecary. Danielle and Toni both work extensively with women’s hormonal and metabolic health. You can book a free discovery call to discuss your case and determine the right practitioner for you at nourishingapothecary.com. #HighTestosterone #PCOS #Androgens #WomensHormones #HormoneHealth

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