@juublian.neuro: Neurotoxicity of Trenbolone #pharmacology #neurology #gear #anabolic

Tdd_4717 :

Fearmongering

SergiuEs :

does it really matter tho? alchohol also kills neurons

userr.5oh :

steph :

The fact that it cannot be explained by confounding variables makes me believe it’s all a bunch of BS

Stefan :

Nuh uh

Richinto :

“Oh my goodness…. You’re over simplifying all this… why isn’t this video more in depth… why isn’t it 3 hours and 34 minutes on TikTok…. Oh my goodness dude….”

🧬RPO🧬 :

Yes this is really interesting, I’ve researched this toxicity myself and I’m down to chat about it w u or maybe we can collab if ur down

Julian :

A few notes, as I’m oversimplifying in the video for the sake of time restrictions: 1. The correct term would be that AAS “atrophy” the brain, not rot it. Just said rot for clickbait. If your brain was literally rotting then we’d lowkey have a bigger problem 2. The first cited study uses quite high concentrations of trenbolone, which may not translate directly to human application. Note here that due to lipophilicity, neuronal concentration of AAS is likely going to be higher than blood serum concentration in an organism. 3. Tubb3 stands for Tubulin beta-3 chain. It is responsible for other things beyond neurite outgrowth (downstream) via betaIII-tubulin, but its primary role is to help form microtubules which play large roles in axonal / dendritic guidance.

GOAD :

how to counteract?

🕋☪️SCI SEAN LOVES POOP👳‍♂️ :

Bro no 💀💀💀

drain :

Yes, my brain is definitely suffering from this compound.

core :

The dose makes the poison. As with the vast majority of pharmacologically active compounds, pharmacodynamics change according to concentration, and often substantially so. It cannot be assumed with any confidence that low-moderate doses of Trenbolone inhibit Tubb3 to any meaningful extent, or even at all.

henry324201 :

You don’t know that it was explicitly the tren that did it and not the horrible sleep quality

. :

@Pear how do i @your mom