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@f4ke_hxn: Perdóname mami 😞pero estaba potente las trampas de seungmin @TikTok @Stray Kids #hanni🐶🐰💞 #paratiiiiiiiiiiiiiiiiiiiiiiiiiiiiiii #flypシ #noflop #nomedejenenflop

Hanni!!🐶🐰💞
Hanni!!🐶🐰💞
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Tuesday 15 September 2026 01:33:12 GMT
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Sindy ❤️ :
2026-09-15 04:36:12
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ကိုယ့်စကားကိုယ်တည်ကြပါ😁😁🙂‍↔️#fyppppppppppppppppppppppp #foryoupage #မင်းတို့idolဖစ်ချင်လို့🤭💖
ကိုယ့်စကားကိုယ်တည်ကြပါ😁😁🙂‍↔️#fyppppppppppppppppppppppp #foryoupage #မင်းတို့idolဖစ်ချင်လို့🤭💖
دعاء النبي لسداد الدين وتوسيع الرزق ♥️
دعاء النبي لسداد الدين وتوسيع الرزق ♥️
hEDS has traditionally been classified as a connective tissue disorder defined by joint hypermobility. This framing never fully explained why hEDS so often comes with immune dysfunction, dysautonomia, and chronic fatigue. New genetic research is reframing it as a multi-system condition. A 2025 study by Gensemer et al. in iScience (MUSC, Norris lab) performed whole-exome sequencing on 200 hEDS patients and identified a recurrent variant in KLK15, a gene active in both connective tissue and immune tissue. A knock-in mouse with the variant developed connective tissue abnormalities in tendons and heart valves alongside dysregulated cytokine signaling. A separate 2026 study by Shirvani et al. in Genes found hEDS-associated variants enriched across three systems: collagen biosynthesis, HLA/immune pathways, and mitochondrial function. KLK15 is described as a contributor, not a confirmed single cause. The variant was found in roughly a third of patients, and hEDS remains genetically complex. This research does not yet enable a genetic test for hEDS, and the 2017 clinical criteria remain the standard for diagnosis. The multi-system genetic paper used machine learning on a single cohort and needs replication. I’m not a doctor, just sharing research I find interesting. Sources: Gensemer et al. (2025). iScience. DOI: 10.1016/j.isci.2025.113343 Shirvani et al. (2026). Genes. DOI: 10.3390/genes17020211 #heds #ehlersdanlos #chronicillness #hypermobileehlersdanlossyndrome
hEDS has traditionally been classified as a connective tissue disorder defined by joint hypermobility. This framing never fully explained why hEDS so often comes with immune dysfunction, dysautonomia, and chronic fatigue. New genetic research is reframing it as a multi-system condition. A 2025 study by Gensemer et al. in iScience (MUSC, Norris lab) performed whole-exome sequencing on 200 hEDS patients and identified a recurrent variant in KLK15, a gene active in both connective tissue and immune tissue. A knock-in mouse with the variant developed connective tissue abnormalities in tendons and heart valves alongside dysregulated cytokine signaling. A separate 2026 study by Shirvani et al. in Genes found hEDS-associated variants enriched across three systems: collagen biosynthesis, HLA/immune pathways, and mitochondrial function. KLK15 is described as a contributor, not a confirmed single cause. The variant was found in roughly a third of patients, and hEDS remains genetically complex. This research does not yet enable a genetic test for hEDS, and the 2017 clinical criteria remain the standard for diagnosis. The multi-system genetic paper used machine learning on a single cohort and needs replication. I’m not a doctor, just sharing research I find interesting. Sources: Gensemer et al. (2025). iScience. DOI: 10.1016/j.isci.2025.113343 Shirvani et al. (2026). Genes. DOI: 10.3390/genes17020211 #heds #ehlersdanlos #chronicillness #hypermobileehlersdanlossyndrome
MUNAFIK: MELAWAN IBLIS mengisah ustad Adam, si spesialis ruqyah, dikenal mampu membantu orang-orang yang diganggu makhluk gaib. Namun semuanya berubah setelah kecelakaan maut merenggut Aini, istrinya, tepat sepulang ia menyelamatkan seorang anak dari gangguan Iblis. Trauma membuat ustad Adam berhenti meruqyah dan memilih mengubur masa lalunya. ‎Sampai Anwar datang meminta bantuannya untuk menyelamatkan Fitri, seorang perawat sekaligus putri Haji Mansur yang merupakan tokoh paling disegani di desa. Awalnya Adam menolak. Tapi pesan terakhir Aini membuatnya kembali terjun ke dunia ruqyah. ‎Masalahnya, gangguan yang dialami Fitri ternyata bukan sekadar teror gaib. Ada rahasia gelap yang disembunyikan keluarga Mansur. Dan semakin Adam menggali, semakin jelas bahwa semua ini mungkin berkaitan dengan masa lalunya sendiri. #munafikmelawaniblis #munafiknyaindo #filmmunafik #bahayajadimunafik #munafiktk01
MUNAFIK: MELAWAN IBLIS mengisah ustad Adam, si spesialis ruqyah, dikenal mampu membantu orang-orang yang diganggu makhluk gaib. Namun semuanya berubah setelah kecelakaan maut merenggut Aini, istrinya, tepat sepulang ia menyelamatkan seorang anak dari gangguan Iblis. Trauma membuat ustad Adam berhenti meruqyah dan memilih mengubur masa lalunya. ‎Sampai Anwar datang meminta bantuannya untuk menyelamatkan Fitri, seorang perawat sekaligus putri Haji Mansur yang merupakan tokoh paling disegani di desa. Awalnya Adam menolak. Tapi pesan terakhir Aini membuatnya kembali terjun ke dunia ruqyah. ‎Masalahnya, gangguan yang dialami Fitri ternyata bukan sekadar teror gaib. Ada rahasia gelap yang disembunyikan keluarga Mansur. Dan semakin Adam menggali, semakin jelas bahwa semua ini mungkin berkaitan dengan masa lalunya sendiri. #munafikmelawaniblis #munafiknyaindo #filmmunafik #bahayajadimunafik #munafiktk01
SELAGI ADA LANGSUNG BUNGKUS #jualbelimotorsecond #bren6jayamotor #6jayamotormalang
SELAGI ADA LANGSUNG BUNGKUS #jualbelimotorsecond #bren6jayamotor #6jayamotormalang

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