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@soleildusud12:

Soleildusud
Soleildusud
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Region: BE
Sunday 01 November 2026 00:08:14 GMT
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actonplace
Agatha❤️ :
Body is what 🔥🔥
2026-11-01 00:12:07
0
aminafashionadjana
ADJA ANA PDG FIEDA BRUXELLES :
fa dionguoma
2026-11-01 09:34:01
1
theongom
THÉO NGOM (artiste-peintre) :
yow khana équipe bleue bleue bou pére nga bok..???
2026-11-01 00:24:27
1
amathlastcri
THE BLADE :
💙
2026-11-01 03:52:37
1
bayefal818
💫jeune 💫pauvre ✌️🇸🇳 :
🥰🥰🥰
2026-11-01 10:55:28
0
amadouousmaneba3
amadouousmaneba3 :
🥰🥰🥰
2026-11-01 01:27:31
0
anta_treccine
Anta LR :
❤️❤️❤️
2026-11-01 10:19:19
0
layegueyebadji
Laye Gueye Badji :
♥️
2026-11-01 06:28:26
0
dabasarr315
Diamant Noir sarr315 :
💕💕💕💕💕🥰🥰🥰🥰🥰
2026-11-01 09:25:44
0
mame.saliou.seul6
Mame saliou :
🥰🥰🥰
2026-11-01 00:12:37
0
amadouousmaneba3
amadouousmaneba3 :
💚💚💚
2026-11-01 01:27:33
0
chernorjalloh6183
Cee jay :
[Rose][Rose][Rose]
2026-11-01 09:35:44
0
siley.ka29
Siley Ka :
❤️❤️❤️
2026-11-01 00:41:14
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user68390219959610
niwe :
🙏🙏🙏
2026-11-01 00:14:45
0
mamadou.sow20062
Mamadou Sow :
🥰🥰🥰
2026-11-01 00:13:22
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nonstress221
🇲🇫🇲🇫🇲🇫❤️ :
🥰🥰🥰🥰🥰🥰🥰🥰🥰❤️❤️❤️❤️❤️❤️❤️❤️❤️❤️
2026-11-01 11:12:49
0
mbayeka275
Mbaye Ka :
🥰🥰🥰
2026-11-01 12:23:51
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Helicobacter pylori is one of the most common causes of chronic gastritis worldwide, and its effects may extend beyond the stomach. By reducing gastric acid secretion, H. pylori infection can impair the absorption of oral levothyroxine, which needs an acidic environment to dissolve and be absorbed properly. This may explain why some patients with hypothyroidism fail to reach normal thyrotropin (TSH) levels even on high doses of thyroxine. Bugdaci et al. (Helicobacter, 2011) studied whether eradicating H. pylori improves thyroid function in hypothyroid patients who had not responded to high-dose thyroxine therapy. The study included hypothyroid patients seen at the Endocrinology and Gastroenterohepatology Clinics of Sisli Etfal Training and Research Hospital between 2009 and 2010 who had not responded to high doses of thyroxine. All patients underwent upper GI endoscopy with duodenal, antral, and corporal biopsies and jejunal sampling, and only those without intestinal pathology were included. Thyroid function was measured twice before and after H. pylori eradication. After eradication, mean TSH fell sharply from 30.5 ± 28.8 to 4.2 ± 10.6 IU/mL (p < .001). Free T3 rose from 2.64 ± 0.56 to 3.02 ± 0.61 pg/mL (p = .002), and free T4 rose from 0.92 ± 0.32 to 1.3 ± 0.34 ng/mL (p < .001). TSH decreased in every patient, but 21% developed factitious thyrotoxicosis. The authors concluded that H. pylori gastritis may underlie poor response to thyroxine. They also warned that eradicating the infection in patients on high thyroxine doses carries a risk of over-replacement, so thyroid function should be monitored closely after treatment and the dose adjusted. disclaimer educational purposes only medical knowledge
Helicobacter pylori is one of the most common causes of chronic gastritis worldwide, and its effects may extend beyond the stomach. By reducing gastric acid secretion, H. pylori infection can impair the absorption of oral levothyroxine, which needs an acidic environment to dissolve and be absorbed properly. This may explain why some patients with hypothyroidism fail to reach normal thyrotropin (TSH) levels even on high doses of thyroxine. Bugdaci et al. (Helicobacter, 2011) studied whether eradicating H. pylori improves thyroid function in hypothyroid patients who had not responded to high-dose thyroxine therapy. The study included hypothyroid patients seen at the Endocrinology and Gastroenterohepatology Clinics of Sisli Etfal Training and Research Hospital between 2009 and 2010 who had not responded to high doses of thyroxine. All patients underwent upper GI endoscopy with duodenal, antral, and corporal biopsies and jejunal sampling, and only those without intestinal pathology were included. Thyroid function was measured twice before and after H. pylori eradication. After eradication, mean TSH fell sharply from 30.5 ± 28.8 to 4.2 ± 10.6 IU/mL (p < .001). Free T3 rose from 2.64 ± 0.56 to 3.02 ± 0.61 pg/mL (p = .002), and free T4 rose from 0.92 ± 0.32 to 1.3 ± 0.34 ng/mL (p < .001). TSH decreased in every patient, but 21% developed factitious thyrotoxicosis. The authors concluded that H. pylori gastritis may underlie poor response to thyroxine. They also warned that eradicating the infection in patients on high thyroxine doses carries a risk of over-replacement, so thyroid function should be monitored closely after treatment and the dose adjusted. disclaimer educational purposes only medical knowledge

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