@meiimei2x: Lụm về mặc liền

Meii Thập Cẩm ♈️
Meii Thập Cẩm ♈️
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Thursday 31 July 2025 13:57:08 GMT
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ilovebicep
ilovebicep :
Link đâu ạ
2025-09-28 08:17:12
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tngcz98
Freeman :
vai rộng bắp tay to mặc ổn ko ạ
2025-09-22 11:44:16
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vohuynhdongthi
Đông Thi :
Thi: Mẫu mặc sz nào v ạ
2025-09-09 08:11:58
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u.dytwng
TDy 🦢ྀི :
Em vai rộng mặc dô ổn ko ạ
2025-08-07 17:51:13
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meiimei2x
Meii Thập Cẩm ♈️ :
Dạng thun á
2025-07-31 14:06:04
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Hyperparathyroidism (Overactive parathyroids → ↑ PTH) Types Primary – usually due to a benign adenoma, hyperplasia, or (rarely) carcinoma. Secondary – compensatory increase in PTH due to chronic low calcium (e.g., chronic kidney disease, vitamin D deficiency). Tertiary – autonomous PTH secretion after long-standing secondary hyperparathyroidism (often in renal patients). Effects High PTH → High calcium (hypercalcemia) + Low phosphate (hypophosphatemia) Bones, stones, abdominal groans, psychic moans. Bone pain, osteoporosis, fractures. Kidney stones, polyuria.. GI issues: constipation, peptic ulcers, pancreatitis. Neuro: fatigue, depression, confusion Management Primary: Surgery (parathyroidectomy) if symptomatic or severe; otherwise monitor. Secondary: Treat underlying cause (e.g., phosphate binders, vitamin D, dialysis). Hydration, bisphosphonates, calcimimetics (cinacalcet) may help. Hypoparathyroidism (Underactive parathyroids → ↓ PTH) Causes Most common: post-surgical (thyroid/parathyroid surgery). Autoimmune destruction. Genetic syndromes (DiGeorge syndrome). Low magnesium (needed for PTH secretion Hypoparathyroidism (Underactive parathyroids → Effects Low PTH → Low calcium (hypocalcemia) + High phosphate (hyperphosphatemia) Symptoms of hypocalcemia: Tingling around mouth, hands, feet Muscle cramps, spasms (tetany), carpopedal spasm Chvostek’s sign (facial spasm when tapping cheek) Trousseau’s sign (carpal spasm with BP cuff inflation) Severe: seizures, cardiac arrhythmias).
Hyperparathyroidism (Overactive parathyroids → ↑ PTH) Types Primary – usually due to a benign adenoma, hyperplasia, or (rarely) carcinoma. Secondary – compensatory increase in PTH due to chronic low calcium (e.g., chronic kidney disease, vitamin D deficiency). Tertiary – autonomous PTH secretion after long-standing secondary hyperparathyroidism (often in renal patients). Effects High PTH → High calcium (hypercalcemia) + Low phosphate (hypophosphatemia) Bones, stones, abdominal groans, psychic moans. Bone pain, osteoporosis, fractures. Kidney stones, polyuria.. GI issues: constipation, peptic ulcers, pancreatitis. Neuro: fatigue, depression, confusion Management Primary: Surgery (parathyroidectomy) if symptomatic or severe; otherwise monitor. Secondary: Treat underlying cause (e.g., phosphate binders, vitamin D, dialysis). Hydration, bisphosphonates, calcimimetics (cinacalcet) may help. Hypoparathyroidism (Underactive parathyroids → ↓ PTH) Causes Most common: post-surgical (thyroid/parathyroid surgery). Autoimmune destruction. Genetic syndromes (DiGeorge syndrome). Low magnesium (needed for PTH secretion Hypoparathyroidism (Underactive parathyroids → Effects Low PTH → Low calcium (hypocalcemia) + High phosphate (hyperphosphatemia) Symptoms of hypocalcemia: Tingling around mouth, hands, feet Muscle cramps, spasms (tetany), carpopedal spasm Chvostek’s sign (facial spasm when tapping cheek) Trousseau’s sign (carpal spasm with BP cuff inflation) Severe: seizures, cardiac arrhythmias).

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